Man it's so fucked up that this person said god bless idiopathic pulmonary fibrosis.
@Telegraph: In 2020, life expectancy for people with cystic fibrosis was just 36. A new drug available on the NHS may be about to change that Find out more 👇
Also i might just do a birthday donothon, or ill just ask some people to donate to the Cystic Fibrosis foundation
(BioRxiv All) Systemic effects of cystic fibrosis transmembrane conductance regulator (CFTR) modulators on the blood proteome: Cystic fibrosis (CF), resulting from a dysfunction in the cystic fibrosis transmembrane conductance regulator (CFTR), affects… http://dlvr.it/TFVnXg#MassSpecRSS
Systemic effects of cystic fibrosis transmembrane conductance regulator (CFTR) modulators on the blood proteome www.biorxiv.org/content/10.1101/2024.10.18.619058v1
Cystic fibrosis (CF), resulting from a dysfunction in the cystic fibrosis transmembrane conductance
Systemic effects of cystic fibrosis transmembrane conductance regulator (CFTR) modulators on the blood proteome www.biorxiv.org/content/10.1101/2024.10.18.619058v1
Cystic fibrosis (CF), resulting from a dysfunction in the cystic fibrosis transmembrane conductance
GSK and Boston scientists team up to study pulmonary fibrosis and develop new treatments www.fiercebiotech.com/research/gsk... The collaborators will also work to develop new organoid models of lung disease. By combining lung cells and mesenchymal cells, which are stem cells that can turn into 1/
London-based GSK is crossing the pond to form a new lung disease research collaboration with Boston scientists. | London-based GSK is crossing the pond to form a new lung disease research collaboratio...
is an inspiring strategy to promote normal regeneration and anti-fibrosis, and TRβ activation is a highly promising strategy to fulfill this intractable task by orchestrating KLF2 and CEBPA. 2/2
TRβ activation confers AT2-to-AT1 cell differentiation and anti-fibrosis during lung repair via KLF2 and CEBPA nature.com/articles/s41... In conclusion, our observations support the idea that masking pathologic plasticity and facilitating endogenous differentiation potential of AT2 cells 1/2
Here, Xin Pan and colleagues found that activation of thyroid hormone receptor-β is necessary for alveolar epithelial cell differentiation during lung repair and regeneration, and confirmed a specific...